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the money, but it is a reality nonetheless Again, this is driven by supply and demand If you have an abundant supply of option writers away from the market or a weak group of buyers, the implied is likely to retreat If the market appears to have little or no potential for growth or disaster, you will see a similar situation What we can learn from the volatility smile is to spot when we have distortions that we may be able to capitalize on Selling call and put premiums on the outside edges of a steep volatility smile is likely to be a good opportunity to capture the volatility Someone is or has been willing to buy these options at high volatility for the smile to exist so you must be certain of your fundamental and technical analysis You can extrapolate from the smile what strategies might be most effective in each situation You have to reverse your thinking and consider trading implied volatility and not the market fundamental itself If volatility is low in the-at-the-money options and elevated on both ends, we might consider a short out-of-the-money condor, short strangle, long straddle, long at-the-money call or put, bull call or bear put spreads, and ratio spreads covered calls and puts All of these take advantage of out-of-the money implied volatility For elevated implied volatility in the center like the frown, we have another set of trading strategies: a short iron butter y, short straddle, long strangle, back spreads, long out-of-the-money calls or puts, and long condors These trades take advantage of your being able to purchase low out-of-the-money implied volatility in relation to the at-the-money positions Pricing your option trades based on volatility as much as on the price and on market fundamentals will do a number of things for your trading:.

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binski signs, grasp and suck re exes, weakness of the legs progressing to paraplegia, bladder and bowel incontinence re ecting spinal cord or cerebral involvement, and abulia or mutism are prominent in the later stages of the disease In the untreated case, the dementia evolves relatively rapidly, over a period of weeks or months; survival after the onset of dementia is generally 3 to 6 months but may be considerably longer Tests of psychomotor speed seem to be most sensitive in the early stages of dementia (eg, trail-making, pegboard, and symbol-digit testing) Epstein and colleagues have described a similar disorder in children, who develop a progressive encephalopathy as the primary manifestation of AIDS The disease in children is characterized by an impairment of cognitive functions and spastic weakness and secondarily by impairment of brain growth The CSF in patients with AIDS dementia (but lacking other manifestations of AIDS) may be normal or show only a slight elevation of protein content and, less frequently, a mild lymphocytosis HIV can be isolated from the CSF In the CT scan there is widening of the sulci and enlargement of the ventricles; MRI may show patchy but con uent or diffuse white matter changes with illde ned margins (Fig 33-2) These ndings are particularly useful in diagnosis, although CMV infection of the brain in AIDS patients produces a similar MRI appearance, as described further on The pathologic basis of the dementia appears to be a diffuse and multifocal rarefaction of the cerebral white matter, accompanied by scanty perivascular in ltrates of lymphocytes and clusters of a few foamy macrophages, microglial nodules, and multinucleated giant cells (Navia et al) Evidence of CMV infection may be added, but accumulating virologic evidence indicates that the AIDS dementia complex is due to direct infection with HIV Which of these changes, or the cortical atrophy, correspond most closely to the presence and severity of dementia has not been settled The pathologic changes in AIDS dementia are actually not as uniform as portrayed here In one group of patients, there is a diffuse pallor of the cerebral white matter, most obvious with myelin stains, accompanied by reactive astrocytes and macrophages; the myelin pallor seems to re ect a breakdown of the blood-brain barrier In another form of this process, referred to as diffuse poliodystrophy, there is widespread astrocytosis and microglial activation in the.

Adapted by permission from Brew B, Sidtis J, Petito DK, Price RW: The neurologic complications of AIDS and human immunode ciency virus infection, in Plum F (ed), Advances in Contemporary Neurology Philadelphia, Davis, 1988, chap 1

present in this manner, but the frequency is far higher, close to twothirds, after the constitutional symptoms and opportunistic infections of AIDS are established In children with AIDS, dementia is more common than all opportunistic infections, over 60 percent of children eventually being affected This disorder takes the form of a slowly or rapidly progressive dementia (loss of retentive memory, inattentiveness, language disorder, and apathy) accompanied by abnormalities of motor function Patients complain of being unable to follow conversations, taking longer to complete daily tasks, and becoming forgetful Inco-ordination of the limbs, ataxia of gait, and impairment of smooth pursuit and saccadic eye movements are usually early accompaniments of the dementia Heightened tendon re exes, Ba-

This gives you some idea of the linkage of the BW query to the OLAP universe Once this process of creating an OLAP universe is complete, we can step into the Web Intelligence Rich Client and create the report by dragging and dropping the information into the appropriate columns and rows The following illustration shows the initial screen for the Web Intelligence Rich Client Once you start working in this environment, you will find that a number of functions and tasks are similar to those in the BEx Query Designer in terms of formatting and display options All of the components are found in similar navigational processes as the BEx Query Designer either in the right-click context menu or in the top toolbar, where you click and choose what you need to work on

Figure 33-2 MRI of AIDS leukoencephalopathy There are large areas of white matter change that underlie one form of AIDS dementia; cortical atrophy and ventricular enlargement are evident