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the information across a group of analysts to develop additional information to help drive business for their corporation The functionality available with the BOBJ Explorer is not quite the same as the other reporting tools since it is used primarily for analysis and the sorting of data mining information, there are few formatting and context menu functions to work with Accessing BOBJ Explorer is similar to accessing a search engine screen on the Internet, while other reporting tools present you with a selection of reports for execution Most of the basic functions are available, such as generating reports and queries, using charts and graphics to display the information, and some navigational functions, but the primary goal of the Explorer is to allow you to obtain information and answers at the speed of thought Parameters exist to allow the distribution of the data, but over and above that, the information is displayed as is for analysis Using BusinessObjects Explorer, you can query on terabytes of data and generate an answer for a customer immediately while you are on the phone with that customer and not have to get back to them with decisions on product cost, price, and shipping information With this reporting tool, your business analysts can discuss areas of concern and get answers for questions immediately and not have to go through the process of getting a change request from the BW IT department If this reduces the workload on the BW IT group, that is another benefit Because business and power users can obtain information immediately, they can develop their own reports and test the information immediately This requires a user interface that is user friendly and easy to work with, which is a trait of all the BOBJ products

Compiled by J Mohr, L Caplan, D Pessin, P Kistler, and G Duncan at Massachusetts General Hospital and Beth Israel Hospital, Boston Compiled by C M Fisher and R D Adams in an examination of 740 brains during the year 1949 at Mallory Institute of Pathology, Boston City Hospital Hypertensive encephalopathy, cerebral vein thrombosis, meningovascular syphilis, and polyarteritis nodosa

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quently a brain artery becomes occluded when there is no clinical evidence of coronary or peripheral vascular disease Although atheromatosis is known to have its onset in childhood and adolescence, only in the middle and late years of life is it likely to have clinical effects Hypertension, hyperlipidemia, and diabetes greatly aggravate the process As with coronary and peripheral atherosclerosis, individuals with low blood levels of high-density-lipoprotein (HDL) cholesterol and high levels of low-density-lipoprotein (LDL) cholesterol are particularly disposed to cerebral atherosclerosis (Nubiola et al) Long-duration cigarette smoking, an important factor in the genesis of atherosclerosis, decreases both HDL cholesterol and CBF (Ingall et al) Added to these well-established risk factors is a theorized role of an excess of homocystine and an even more speculative role for chronic in ammation or and intracellular infection of uncertain origin within plaques (Chlamydia pneumoniae has been implicated in some studies) Clinical experience indicates that there are families with a predilection for the development of cerebral atherosclerosis, independent of these wellde ned risk factors Obesity is also a risk factor, in part because of its correlation with diabetes There is a tendency for atheromatous plaques to form at branchings and curves of the cerebral arteries The most frequent sites are in the internal carotid artery, at its origin from the common carotid; in the cervical part of the vertebral arteries and at their junction to form the basilar artery; in the stem or at the main bifurcation of the middle cerebral arteries; in the posterior cerebral arteries as they wind around the midbrain; and in the anterior cerebral arteries as they pass anteriorly and curve over the corpus callosum It is rare for the cerebral arteries to develop plaques beyond their rst major branching Also, it is unusual for the cerebellar and ophthalmic arteries to show atheromatous involvement except in conjunction with hypertension The common carotid and vertebral arteries at their origins from the aorta are frequent sites of atheromatous deposits, but because of abundant collateral arterial pathways, occlusions at these sites are not commonly associated with cerebral ischemia The atheromatous lesions develop and grow silently for 20, 30, or more years; only in the event of a thrombotic complication do they become symptomatic Although atheromatous plaques may narrow the lumen of an artery, causing stenosis, complete occlusion is nearly always the consequence of superimposed thrombosis ( atherothrombosis ) In general, the more severe the atheromatosis, the more likely the thrombotic complication, but the two processes do not always run in parallel One patient with only scattered atheromatous plaques may thrombose a vessel, whereas another with marked atherosclerosis may have only a few thrombosed vessels or none at all Atheromatous lesions may regress to some extent under the in uence of diet and certain drugs Hennerici and colleagues followed a series of patients with carotid stenoses for a period of 18 months and observed spontaneous regression in nearly 20 percent of the lesions In the large majority of cases, however, atherosclerosis is a progressive disease Degeneration of the wall of a sclerotic vessel or hemorrhage into the wall (from rupture of the vasa vasorum) may damage the endothelium This is known to occur particularly in the walls of the internal carotid arteries (and the coronary arteries, where it is a main cause of vessel occlusion) The lesion in the atheromatous plaque apparently excites an in ammatory reaction Platelets and brin then adhere to the damaged part of the wall and form delicate, friable clots, or a subintimal atheromatous deposit may slough, spewing crystalline cholesterol emboli into the lumen with result-.