rdlc data matrix

rdlc data matrix

rdlc data matrix

rdlc data matrix

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(pain, burning, and impaired sensation over ipsilateral half of the face); nucleus and tractus solitarius (loss of taste); and rarely cuneate and gracile nuclei (numbness of ipsilateral limbs) Fragmentary syndromes are more frequent at the onset of the stroke; the fragmentary syndromes are vertigo and ptosis, toppling and vertical diplopia, hoarseness and disequilibrium, etc Vertigo alone, however, is not an indication of lateral medullary infarction The small-

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est infarction we have studied gave rise only to symptoms of lateropulsion and mild ipsilateral limb ataxia The eye signs of lateral medullary infarction may be dif cult to interpret Often there is a fragment of an internuclear ophthalmoplegia or a skew deviation (the globe on the affected side usually higher) Direction changing nystagmus (with different head positions) is said to be the most useful feature that distinguishes labyrinthine disease from brainstem forms of nystagmus, but infarction of the vestibular nucleus as part of the lateral medullary syndrome may also show this sign (see also Chap 15) The entire lateral medullary syndrome, one of the most striking in neurology, is almost always due to infarction, with only a small number of cases being the result of hemorrhage or tumor Although it was traditionally attributed to occlusion in the course of the posterior inferior cerebellar artery (PICA), careful studies have shown that in 8 out of 10 cases it is the vertebral artery that is occluded by atherothrombosis; in the remainder, either the posterior inferior cerebellar artery or one of the lateral medullary arteries is occluded Embolism is a less frequent cause The inferior cerebellum is usually affected, causing vomiting, vertigo, and ataxia In recent years, we have become aware from our own patients that although most of those with lateral medullary infarction do well and have considerable recovery, sudden and unexpected death may occur from respiratory or cardiac arrest, sometimes even in the absence of cerebellar swelling or basilar artery thrombosis Cases of this nature have been reviewed by Norving and Cronqvist The related and important issue of cerebellar swelling after vertebral artery or PICA occlusion and the subsequent need for surgery is discussed under Treatment of Cerebral Edema and Raised Intracranial Pressure, later in the chapter Basilar Artery The branches of the basilar artery may be instructively grouped as follows: (1) paramedian, 7 to 10 in number, supplying a wedge of pons on either side of the midline; (2) short circumferential, 5 to 7 in number, supplying the lateral two-thirds of the pons and the middle and superior cerebellar peduncles; (3) long circumferential, 2 on each side (the superior and anterior inferior cerebellar arteries), which run laterally around the pons to reach the cerebellar hemispheres (Figs 34-11 and 34-12); and (4) several paramedian (interpeduncular) branches at the bifurcation of the basilar artery and origins of the posterior cerebral arteries supplying the high midbrain and medial subthalamic regions These interpeduncular and other short proximal branches of the posterior cerebral artery have been described above The picture of basilar artery occlusion due to thrombosis may arise in several ways: (1) occlusion of the basilar artery itself, usually in the lower or middle third at the site of an atherosclerotic plaque and superimposed thrombosis; (2) occlusion of both vertebral arteries; and (3) occlusion of a single vertebral artery when it is the only one of adequate size Also, thrombosis may involve a branch of the basilar artery rather than the trunk (basilar branch occlusion) When there is embolism, the clot usually lodges at the upper bifurcation of the basilar or in one of the posterior cerebral arteries, since the embolus, if it is small enough to pass through the vertebral artery, easily traverses the length of the basilar artery, which is of greater diameter than either vertebral artery The syndrome of basilar artery occlusion, as delineated by Kubik and Adams, re ects the involvement of a large number of structures: corticospinal and corticobulbar tracts, cerebellum, middle and superior cerebellar peduncles, medial and lateral lemnisci, spinothalamic.

As you can see, the SAP menu offers Open Report, Create a New Report from a Query, Start BW Query Designer, and other options, but the goal normally is to develop a formatted report that can be distributed using another component of Crystal Reports Crystal Server to a specific group Normally, these stakeholders are looking for a very nicely formatted report with information that can be shared externally The look and feel of Crystal Reports hasn t changed much since I initially worked with it eight years ago The additional functionality that allows you to use a wizard to start your build and guide you through the

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